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One idea is that some people may clear the virus rapidly, before it reaches detectable levels, due to existing immunity to other coronaviruses like those that cause the common cold.
A fever from a cold tends to be short-lived, if it manifests at all. And unless you have asthma, trouble breathing is a singular symptom of COVID-19, too. “In people with asthma, allergies and ...
The timeline of the COVID-19 pandemic lists the articles containing the chronology and epidemiology of SARS-CoV-2, [1] the virus that causes the coronavirus disease 2019 ( COVID-19) and is responsible for the COVID-19 pandemic . The first human cases of COVID-19 occurred in Wuhan, People's Republic of China, on or about 17 November 2019. [2]
The effects of parasitic worms, or helminths, on the immune system is a recently emerging topic of study among immunologists and other biologists. Experiments have involved a wide range of parasites, diseases, and hosts. The effects on humans have been of special interest. The tendency of many parasitic worms to pacify the host's immune ...
The Lancet COVID-19 Commission task force. In November 2020, Richard Horton, editor of The Lancet, appointed economist Jeffrey Sachs as chair of its COVID-19 Commission, with wide-ranging goals relating to the virus and pandemic. Sachs set up a number of task forces, including one on the origins of the virus.
Common cold. The common cold or the cold is a viral infectious disease of the upper respiratory tract that primarily affects the respiratory mucosa of the nose, throat, sinuses, and larynx. [6] [8] Signs and symptoms may appear in as little as two days after exposure to the virus. [6] These may include coughing, sore throat, runny nose ...
The transmission of COVID-19 is the passing of coronavirus disease 2019 from person to person. COVID-19 is mainly transmitted when people breathe in air contaminated by droplets / aerosols and small airborne particles containing the virus. Infected people exhale those particles as they breathe, talk, cough, sneeze, or sing.
Antibody-dependent enhancement ( ADE ), sometimes less precisely called immune enhancement or disease enhancement, is a phenomenon in which binding of a virus to suboptimal antibodies enhances its entry into host cells, followed by its replication. [1] [2] The suboptimal antibodies can result from natural infection or from vaccination.